• No results found

DNA repair and gene targeting in plant end-joining mutants Jia, Q.

N/A
N/A
Protected

Academic year: 2021

Share "DNA repair and gene targeting in plant end-joining mutants Jia, Q."

Copied!
2
0
0

Bezig met laden.... (Bekijk nu de volledige tekst)

Hele tekst

(1)

DNA repair and gene targeting in plant end-joining mutants

Jia, Q.

Citation

Jia, Q. (2011, April 21). DNA repair and gene targeting in plant end-joining mutants. Retrieved from https://hdl.handle.net/1887/17582

Version: Corrected Publisher’s Version

License: Licence agreement concerning inclusion of doctoral thesis in the Institutional Repository of the University of Leiden Downloaded from: https://hdl.handle.net/1887/17582

Note: To cite this publication please use the final published version (if applicable).

(2)

137

cv

Curriculum Vitae

Qi Jia was born on March 25th, 1981 in Wuhu, Anhui Province, China. She attended high school at Wuhu No. 1 middle school. In 1998, she started studies at the Biotechnology Department of Shenyang Agricultural University, China.

In 1999, she started her research training at the Enzyme Engineering Lab under the supervision of Dr. Hongman Chen and Dr. Guoshi Kan with the project on the usage of chitinase to protect the tomato from diseases and in 2002 she got her bachelor degree in biotechnology. After her graduation, she worked as an editor for half a year in Wuhu. From 2003 to 2006, she was a master student of biochemistry and molecular biology in Nanjing University in China, and did research in the National Key Laboratory of Pharmaceutical Biotechnology under the supervisor of Prof. Dr. Zichun Hua. During this period, she worked on the one hand on the characterization of tomato Senu3 and senescence in tomato, and on the other hand on expression, purifi cation and analysis of the recombinant proteins thymosin beta 4 and thymosin beta 10 in E. coli. She obtained her MSc degree in 2006, and in the same year she started her PhD project at the Molecular and Development Genetics department, Institute of Biology, Leiden University (IBL) in the Netherlands, under the supervision of Dr. B. Sylvia de Pater and Prof. Dr. Paul J.J. Hooykaas. Th e topic of her project was DNA repair and gene targeting in end joining plant mutants (this thesis). During this time she was also involved in research leading to a publication about the role of Arabidopsis eIF4B2 in PCD (Gaussand, Jia, van der Graaff and et al, Frontier in Plant Physiology, 2011).

Referenties

GERELATEERDE DOCUMENTEN

License: Licence agreement concerning inclusion of doctoral thesis in the Institutional Repository of the University of Leiden Downloaded from: https://hdl.handle.net/1887/17582..

Non-homologous end joining (NHEJ) is the predominant pathway for the repair of DNA double strand breaks in higher eukaryotes, such as plants and mammals.. Th is pathway has

In order to study the role of AtKu80, AtKu70 and AtLig4 in Agrobacterium transformation and gene targeting, Arabidopsis thaliana (ecotype Col-0) homozygous T-DNA

To confi rm this hypothesis, RNA expression levels of AtParp2 and some other genes involved in DSB repair was analyzed using Q-RT-PCR in the C-NHEJ mutants (Atku70, Atku80

T-DNA integration and gene targeting in the Atku80, Atp1p2 and Atp1p2k80 mutants Double strand break repair mechanisms are hypothesized to control the integration of

In order to further analyze its role in DNA repair, we isolated the Atlig6 and Atlig4lig6 double mutants and determined the eff ects of the mutations on T-DNA integration

Th e Atparp1parp2ku80 (Atp1p2k80) triple mutant was hypersensitive as compared to the individual mutants to agents generating DSBs (and SSBs), indicating that the AtParp

Er kan dus worden geconcludeerd dat planten een aantal NHEJ routes hebben om DSB-en te herstellen, niet alleen de klassieke Ku-afhankelijke route, maar ook