Role of reactive oxygen species in rheumatoid arthritis synovial T lymphocytes

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Role of reactive oxygen species in rheumatoid arthritis synovial T

lymphocytes

Remans, Philip Herman Jozef

Citation

Remans, P. H. J. (2006, September 12). Role of reactive oxygen species in rheumatoid arthritis synovial T lymphocytes. Retrieved from https://hdl.handle.net/1887/4569 Version: Corrected Publisher’s Version

License: Licence agreement concerning inclusion of doctoral thesis in the_{Institutional Repository of the University of Leiden} Downloaded from: https://hdl.handle.net/1887/4569

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Reference list

1. Evidence for the role of an altered redox state in hyporesponsiveness of synovial T cells in rheumatoid arthritis. Maurice MM, Nakamura H, van der Voort EA, van Vliet AI, Staal FJ, Tak PP, Breedveld FC, Verweij CL. J Immunol. 1997; 158:1458-65.

2. Displacement of linker for activation of T cells from the plasma membrane due to redox balance alterations results in hyporesponsiveness of synovial fluid T lymphocytes in rheumatoid arthritis. Gringhuis SI, Leow A, Papendrecht-Van Der Voort EA, Remans PH, Breedveld FC, Verweij CL. J Immunol. 2000; 164:2170-9.

3. Cope AP. Studies of T-cell activation in chronic inflammation. Arthritis Res 2002; 4:S197-S211.

4. Bowie A, O'Neill LA. Oxidative stress and nuclear factor-kappaB activation: a

reassessment of the evidence in the light of recent discoveries. Biochem Pharmacol 2000; 59:13-23.

5. Li N, Karin M. Is NF-kappaB the sensor of oxidative stress? FASEB J 1999; 13:1137-43. 6. Collantes E, Valle BM, Mazorra V, Macho A, Aranda E, Munoz E. Nuclear factor-kappa

B activity in T cells from patients with rheumatic diseases: a preliminary report. Ann Rheum Dis 1998; 57(12):738-41.

7. De Rosa SC, Zaretsky MD, Dubs JG, Roederer M, Anderson M, Green A, Mitra D, Watanabe N, Nakamura H, Tjioe I, Deresinski SC, Moore WA, Ela SW, Parks D, Herzenberg LA, Herzenberg LA. N-acetylcysteine replenishes glutathione in HIV infection. Eur J Clin Invest. 2000; 30:915-29.

8. Sochman J. N-acetylcysteine in acute cardiology: 10 years later: what do we know and what would we like to know?! J Am Coll Cardiol. 2002; 39:1422-8.

9. Large-dose ascorbic acid administration suppresses the development of arthritis in adjuvant-infected rats. Sakai A, Hirano T, Okazaki R, Okimoto N, Tanaka K, Nakamura T. Arch Orthop Trauma Surg. 1999; 119:121-6

10. Biochemical changes in arthritic rats under the influence of vitamin E. Kheir-Eldin AA, Hamdy MA, Motawi TK, Shaheen AA, Abd el Gawad HM Agents Actions. 1992; 36:300-5.