• No results found

Release characteristics of cardiac proteins after reversible or irreversible myocardial damage

N/A
N/A
Protected

Academic year: 2021

Share "Release characteristics of cardiac proteins after reversible or irreversible myocardial damage"

Copied!
5
0
0

Bezig met laden.... (Bekijk nu de volledige tekst)

Hele tekst

(1)

Release characteristics of cardiac proteins after reversible or irreversible myocardial damage

Hessel, M.H.M.

Citation

Hessel, M. H. M. (2008, February 7). Release characteristics of cardiac proteins after reversible or irreversible myocardial damage. Retrieved from

https://hdl.handle.net/1887/12593

Version: Corrected Publisher’s Version

License: Licence agreement concerning inclusion of doctoral thesis in the Institutional Repository of the University of Leiden

Downloaded from: https://hdl.handle.net/1887/12593

Note: To cite this publication please use the final published version (if applicable).

(2)

List of publications

(3)

196

(4)

List of publications

197

List of publications

L. Li#, M.H.M. Hessel#, E.J.M. van der Valk, W.H. Bax, I. van der Linden, and A. van der Laarse. Partial and delayed release of troponin-I compared with the release of lactate dehydrogenase from necrotic cardiomyocytes. Pflügers Archiv- Eur J Physiol 2004; 448: 146-152. (# both authors contributed equally).

M.H.M. Hessel, P. Steendijk, B. den Adel, C.I. Schutte, and A. van der Laarse. Characterization of right ventricular function after monocrotaline-induced pulmonary hypertension in the intact rat. Am J Physiol Heart Circ Physiol 2006; 291: H2424-H2430.

M.H.M. Hessel, G.B. Bleeker, J.J. Bax, M.M. Henneman, B. den Adel, M. Klok, D.E. Atsma, and A. van der Laarse. Reverse ventricular remodeling after cardiac resynchronization therapy is associated with a reduction in serum tenascin-C and plasma matrix metalloproteinase-9 levels. Eur J Heart Failure 2007; 9 (10):1058-1063.

E.C.H.J. Michielsen, M.H.M. Hessel, A. van der Laarse, W. Wodzig, and M.P. van Dieijen-Visser.

Heterogeniteit van circulerend cardiaal troponin T. Ned Tijdschr Klin Chem 2007; 32:192-194.

M.H.M. Hessel, D.E. Atsma, E.J.M. van der Valk, W.H. Bax, M.J. Schalij, and A. van der Laarse. Release of cardiac troponin I from viable cardiomyocytes is mediated by integrin stimulation. Pflügers Archiv- Eur J Physiol 2007; in press.

S. Umar, M.H.M. Hessel, P. Steendijk, W.H. Bax, C.I. Schutte, M. J. Schalij, E.E. van der Wall, D.E.

Atsma, and A. van der Laarse. Activation of signaling molecules and matrix metalloproteinases in right ventricular myocardium of rats with pulmonary hypertension. Pathol Res Pract 2007; 203: 869-872.

M.H.M. Hessel, P. Steendijk, B. den Adel, C.I. Schutte and A. van der Laarse. Pressure overload-induced right ventricular dilatation is associated with re-expression of myocardial tenascin-C and elevated plasma levels of tenascin-C. Submitted.

M.H.M. Hessel, E.C.H.J. Michielsen, D.E. Atsma, M.J. Schalij, E.J.M. van der Valk, W.H. Bax, W.T.

Hermens, M.P. van Dieijen-Visser, and A. van der Laarse. Release kinetics of intact and degraded cardiac troponin I and T after irreversible cardiomyocyte damage. Submitted.

(5)

198

Referenties

GERELATEERDE DOCUMENTEN

was to characterize the release kinetics of intact cTnI, intact cTnT, and degradation products of cTnI and cTnT, in relation to LDH, the golden standard for cell

The purpose of the present study is therefore to investigate whether (1) RGD-induced integrin stimulation causes release of intact and/or fragmented cTnI

We hypothesize that patients with congestive heart failure (CHF) with elevated serum cTnT levels at baseline had myocardial cTnT release due to myocardial

CO and stroke volume were maintained, indicating a compensatory state at the expense of increased RV end-diastolic and end-systolic volumes and increased RV

MCT-induced RV dilatation in rats with heart failure (MCT80) was associated with a significant upregulation of TNC gene expression, resulting in re- expression of

Methods: Clinical parameters, left ventricular (LV) volumes, and circulating levels of tenascin-C (TNC), matrix metalloproteinase-2 (MMP-2), MMP-9, and amino-terminal

However, reverse ventricular remodeling is associated with a reduction in plasma MMP9 levels, indicating that plasma MMP9 can be used as a marker of reverse ventricular

Om de rol van serum TNC, MMP2 en MMP9 concentraties als biomarkers voor progressieve kamerdilatatie te kunnen bestuderen in patiënten, hebben we TNC, MMP2 en