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RAGE and the innate immune response in infection and inflammation
van Zoelen, M.A.D.
Publication date 2009
Link to publication
Citation for published version (APA):
van Zoelen, M. A. D. (2009). RAGE and the innate immune response in infection and inflammation.
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Chapter 1
121
141
ContEnts
1. General introduction and outline of the thesis 9
Part I: RAGE and infection
2. Receptor for advanced glycation end products facilitates host
defense during Escherichia coli induced abdominal sepsis in mice. 23
Journal of Infectious Diseases, 2009
3. The receptor for advanced glycation end products impairs host defense in
pneumococcal pneumonia. 43
Journal of Immunology, 2009
4. Receptor for advanced glycation end products protects against
Klebsiella pneumoniae induced pneumonia in mice. 67
Submitted
5. Receptor for advanced glycation end products is protective
during murine tuberculosis. 85
Submitted
6. Receptor for advanced glycation end products is detrimental during
influenza A virus pneumonia. 101
Virology, 2009
Part II: soluble RAGE, RAGE ligands and damage-
associated molecular patterns
7. Ligands of the receptor for advanced glycation end products, including high mobility group box (HMGB) 1, limit bacterial dissemination during
Escherichia coli peritonitis. Under revision
8. Systemic and local high mobility group box 1 concentrations during severe infection.
9. Pulmonary levels of high mobility group box 1 during mechanical ventilation and ventilator-associated pneumonia.
Shock, 2008
10. Role of Toll-like receptors 2 and 4, and the receptor for advanced glycation end products in high mobility group box 1 induced inflammation in vivo. Shock, 2008
11. Expression and role of myeloid-related protein-14 in clinical and experimental sepsis.
American Journal of Respiratory and Critical Care Medicine, 2009
Part III: soluble RAGE levels during inflammation
12. S100A12 and soluble RAGE levels during severe infection.
Submitted
13. Neutrophil-derived S100A12 in acute lung injury and respiratory distress syndrome.
Critical Care Medicine, 2007
14. Urokinase plasminogen activator receptor deficient mice demonstrate reduced hyperoxia induced lung inury.
American Journal of Pathology, 2009
15. Endogenous monocyte chemoattractant protein-1 promotes lung inflammation induced by LPS and LTA.
Submitted
16. Summary, general discussion and future perspectives for research and therapy
Nederlands samenvatting voor niet-ingewijden Acknowledgements/Dankwoord List of publications List of contributors 159 173 185 207 219 235 253 271 281 287 290 292
