• No results found

Biology of monocyte interactions with the endothelium : the platelet factor - Table of Contents

N/A
N/A
Info
Downloaden
Protected

Academic year: 2021

Share "Biology of monocyte interactions with the endothelium : the platelet factor - Table of Contents"

Copied!
2
0
0

Bezig met laden.... (Bekijk nu de volledige tekst)

Download het nu ( 2 pagina )

Hele tekst

(1)

UvA-DARE is a service provided by the library of the University of Amsterdam (https://dare.uva.nl)

UvA-DARE (Digital Academic Repository)

Biology of monocyte interactions with the endothelium : the platelet factor

da Costa Martins, P.A.

Publication date

2005

Link to publication

Citation for published version (APA):

da Costa Martins, P. A. (2005). Biology of monocyte interactions with the endothelium : the

platelet factor.

General rights

It is not permitted to download or to forward/distribute the text or part of it without the consent of the author(s) and/or copyright holder(s), other than for strictly personal, individual use, unless the work is under an open content license (like Creative Commons).

Disclaimer/Complaints regulations

If you believe that digital publication of certain material infringes any of your rights or (privacy) interests, please let the Library know, stating your reasons. In case of a legitimate complaint, the Library will make the material inaccessible and/or remove it from the website. Please Ask the Library: https://uba.uva.nl/en/contact, or a letter to: Library of the University of Amsterdam, Secretariat, Singel 425, 1012 WP Amsterdam, The Netherlands. You will be contacted as soon as possible.

(2)

Tablee of Contents

Chapterr 1 Introductionn and aim of study 11 1

Chapterr 2 Platelet-monocyte complexes support monocyte adhesionn to endothelium by enhancing secondary tetheringg and cluster formation

AtheriosclerAtherioscler Thromb Vase Biol (2004) 24:193-199

37 7

Chapterr 3 P-selectin Glycoprotein Ligand-1 (PSGL-1) is expressed onn endothelial cells and mediates monocyte adhesion too activated endothelium

SubmittedSubmitted for publication

55 5

Chapterr 4 Platelet binding to monocytes increases the adhesive propertiess of monocytes by upregulating the expression andd functionality of fr and (32 integrins

SubmittedSubmitted for publication

77 7

Chapterr 5 LDL-receptor - related protein regulates (32 integrin

-mediatedd leukocyte adhesion

BloodBlood (2005) 105:170-177

97 7

Chapterr 6 DC-SIGN mediates adhesion and rolling of dendritic cells onn primary human umbilical vein endothelial cells through LewisYY antigen expressed on ICAM-2

ManuscriptManuscript in preparation

119 9

Chapterr 7 Discussion, concluding remarks and future perspectives 139 9

Nederlandsee samenvatting/Sumario em português Acknowledgements/Dankwoord/Agradecimentos s

CurriculumCurriculum vitae

155 5 165 5 169 9

Referenties

Download het nu ( PDF - 2 pagina - 146.96 KB )

GERELATEERDE DOCUMENTEN

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 2 Characterization of pal-1, a common proviral insertion site in murine leukemia virus-induced lymphomas of c-myc and pim-1 transgenic mice

In an attempt to identify new collaborating oncogenes in MoMLV-induced tumors in Eu.-myr transgenic mice, another common insertion site, pal-1 (proviral activation in lymphomas

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 3 Retroviral insertions in the bla1 locus result in long-range activation of the bmi1 proto-oncogene in B cell lymphomas

As much as 2 5 % (10/40) of the lymphomas with proviral insertions near bmil also contain MoMLV integration in the eisl/gfi/pall/evi5 lo-.. Proviral integrations in bmil and

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 4 Bmi-1 collaborates with c-Mye in tumorigenesis by inhibiting c-Myc-induced apoptosis via INK4a/ARF

ink4a-ARF. Bmi-1 inhibits c-Myc-induced apoptosis and strongly enhances proliferation in collaboration with myc in an ink4a- ,4i?F-dcpendent manner. [A) Wild-type MEFs were infected

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 5 Gfil promotes thymocyte selection at different stages and inhibits death by neglect, during negative selection and in common receptor γ-ch

Gfil promotes thymocyte selection at different stages and inhibits death by neglect, during negative selec- tion and in common receptor y-chain deficient mice.. Blanca Scheijen 1

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 6 Chronic Myeloid Leukemia-like syndrome and lymphoblastic T cell tumors in mice transgenic for the transcription factor Gfi1

In the current study flow cytometric analysis was performed on spleen and bone marrow, to determine whether differentiation of the other major hematopoietic lineages

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 7 Enforced expression of Gfil alters craniofacial and tooth morphogenesis and induces osteoblastic neural crest cell tumors

The zinc-finger protein Gfil and its close homologue GfilB have been implicated in regulating myeloid and T cell development (Schmidt et al. Here we report that enforced

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Chapter 8 Eμ-myc and gfil collaborate in T cell lymphomagenesis by targeting a p53- and Bcl-2-independent apoptosis pathway

To assess whether enforced gfil expres- sion could affect c-Myc-induced proliferation or inhibit apoptosis in pre-B cells, we analyzed bone marrow cells of wild type, single

Characterization of the Myc collaborating oncogenes Bmi1 and Gfi1 - Summary & Samenvatting

The data described in this thesis indicate that the transcriptional regulators Bmil and Gfil control programmed cell death by different mechanisms in lymphoid cells, but each