• No results found

Microparticles: mediators of cellular and environmental homeostasis - Contents

N/A
N/A
Protected

Academic year: 2021

Share "Microparticles: mediators of cellular and environmental homeostasis - Contents"

Copied!
2
0
0

Bezig met laden.... (Bekijk nu de volledige tekst)

Hele tekst

(1)

UvA-DARE is a service provided by the library of the University of Amsterdam (https://dare.uva.nl)

UvA-DARE (Digital Academic Repository)

Microparticles: mediators of cellular and environmental homeostasis

Böing, A.N.

Publication date

2011

Link to publication

Citation for published version (APA):

Böing, A. N. (2011). Microparticles: mediators of cellular and environmental homeostasis.

General rights

It is not permitted to download or to forward/distribute the text or part of it without the consent of the author(s) and/or copyright holder(s), other than for strictly personal, individual use, unless the work is under an open content license (like Creative Commons).

Disclaimer/Complaints regulations

If you believe that digital publication of certain material infringes any of your rights or (privacy) interests, please let the Library know, stating your reasons. In case of a legitimate complaint, the Library will make the material inaccessible and/or remove it from the website. Please Ask the Library: https://uba.uva.nl/en/contact, or a letter to: Library of the University of Amsterdam, Secretariat, Singel 425, 1012 WP Amsterdam, The Netherlands. You will be contacted as soon as possible.

(2)

Contents

Contents

Chapter 1 Introduction 13

Chapter 2 Inhibition of microparticle release triggers endothelial apoptosis and detachment

Thrombosis and Haemostasis 2007;98:1096-1107

27

Chapter 3 Active caspase-3 is removed from cells by sorting into microparticles

Submitted

53

Chapter 4 Platelet microparticles contain active caspase-3 Platelets 2008;19:96-103

77 Chapter 5 Circulating platelet-derived and placenta-derived microparticles

expose Flt-1 in preeclampsia

Reproductive Sciences 2008;15:1002-1010

93

Chapter 6 Expression of inflammation-related genes in endothelial cells is not directly affected by microparticles from preeclamptic patients Journal of Laboratory and Clinical Medicine 2006;147:310-320

111

Chapter 7 Phospholipid composition of in vitro endothelial microparticles and their in vivo thrombogenic properties

Thrombosis Research 2008;121:865-871

133

Chapter 8 Coagulant tissue factor is not raft associated Submitted

149 Chapter 9 Human alternatively spliced tissue factor is not secreted and does not

trigger coagulation

Journal of Thrombosis and Haemostasis 2009;7:1423-1426

171

Chapter 10 General discussion and summary 179

Chapter 11 Algemene discussie en samenvatting 189

Bibliography 199

Coauthors 201

Curriculum Vitae 203

Referenties

GERELATEERDE DOCUMENTEN

Topographic maps for LI (left) and HI (right), at Pz, for No-Go trials in the Go/No-Go task. Visual representation of one trial in the SSRT. a blue circle) on Go trials (75%

Stopping the “World’s Greatest Threat”: Canadian Policy and Rhetoric towards the Iranian Nuclear Program during Stephen Harper’s Conservative Government, 2006-2015.. by

I think joy, and like, you know, just taking care of yourself and the people around you is really important to this work because we’re in it for the long haul. we have to take care

German’s report, the Corporate Registration program plays a key role in GPEB’s regulatory framework, and is a principal mechanism through which GPEB maintains control over

Britton (1997) concluded that race and gender are contributing factors in stress and further identified the need for future researchers to explore how these intersections

These structural investigations into the mechanism for germ-line antibody recognition of carbohydrate antigens utilizing chlamydial-specific and anti-lipid A antibodies

Our structural analysis revealed that while the N- terminal region of TbFam50.360 adopted a three-helical structure similar to previously characterized trypanosome surface

I showed that data on the structural differences between the native and aggregated forms of the prion protein, obtained from multiple structural proteomics approaches